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Polynucléaires éosinophiles : définition et causes d'un taux élevé
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Granulocyte éosinophile — Wikipédia
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Novel Basophil- or Eosinophil-Depleted Mouse Models for Functional Analyses of Allergic Inflammation
Polynucléaires basophiles bas, élevés : quel est leur rôle ?
PDF) Eosinophils from Physiology to Disease: A Comprehensive Review
Polynucléaires éosinophiles : définition et causes d'un taux élevé
Involvement and therapeutic implications of airway epithelial barrier dysfunction in type 2 inflammation of asthma
Ultrasound and Redox-Triggered Morphology Transitions of Supramolecular Self-assemblies with pH Responsiveness for Triple-Controlled Release | Langmuir
Involvement and therapeutic implications of airway epithelial barrier dysfunction in type 2 inflammation of asthma | Chinese Medical Journal
Involvement and therapeutic implications of airway epithelial barrier dysfunction in type 2 inflammation of asthma
Cancers | Free Full-Text | Re-Expression of Poly/Oligo-Sialylated Adhesion Molecules on the Surface of Tumor Cells Disrupts Their Interaction with Immune-Effector Cells and Contributes to Pathophysiological Immune Escape | HTML
Physiology of blood
References in The β and α2δ auxiliary subunits of voltage-gated calcium channel 1 (Cav1) are required for TH2 lymphocyte function and acute allergic airway inflammation - Journal of Allergy and Clinical Immunology
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Regulation of common neurological disorders by gut microbial metabolites | Experimental & Molecular Medicine
Representative histological appearance of the different cytoplasmic... | Download Scientific Diagram
Mean and single values of IL-33 release in CRSsNP and CRSwNP patients.... | Download Scientific Diagram
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Lipopolysaccharide-Induced Suppression of Airway Th2 Responses Does Not Require IL-12 Production by Dendritic Cells | The Journal of Immunology
Is epithelial barrier hypothesis the key to understanding the higher incidence and excess mortality during COVID‐19 pandemic? The case of Northern Italy - Fiorito - - Allergy - Wiley Online Library
Cancers | Free Full-Text | Re-Expression of Poly/Oligo-Sialylated Adhesion Molecules on the Surface of Tumor Cells Disrupts Their Interaction with Immune-Effector Cells and Contributes to Pathophysiological Immune Escape | HTML